Difference between revisions of "PMID:135755"
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| + | {| id="E50859710214a5" class=" tableEdit PMID_info_table" | ||
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| + | !align=left |Citation | ||
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| + | '''Bassford, PJ Jr, Bradbeer, C , Kadner, RJ and Schnaitman, CA ''' (1976) Transport of vitamin B12 in tonB mutants of Escherichia coli. ''J. Bacteriol.'' '''128''':242-7 | ||
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| + | !align=left |Abstract | ||
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| + | It is known that the tonB mutation in Escherichia coli is responsible for a defect in the transport of iron chelates. These are transported by systems that involve outer membrane components. We found that tonB mutants were also deficient in the secondary, energy-dependent phase of vitamin B12 transport, although the mutants have normal levels of B12 receptors on their cell surface. In addition, tonB mutants derived from vitamin B12 auxotrophs required elevated levels of B12 for normal growth. Maltose uptake, mediated by another transport system involving an outer membrane component, was unaffected by the tonB mutation. | ||
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| + | !align=left |Links | ||
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| + | [http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=135755 PubMed] [http://www.ncbi.nlm.nih.gov/pmc/articles/PMC232849 PMC232849] | ||
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| + | !align=left |Keywords | ||
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| + | Amino Acids/metabolism; Biological Transport, Active; Enterobactin/metabolism; Escherichia coli/growth & development; Escherichia coli/metabolism; Genes; Maltose/metabolism; Mutation; Protein Biosynthesis; Receptors, Drug; Vitamin B 12/metabolism | ||
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| + | ==Main Points of the Paper == | ||
| + | {{LitSignificance}} | ||
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| + | == Materials and Methods Used == | ||
| + | {{LitMaterials}} | ||
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| + | ==Phenotype Annotations== | ||
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| + | !|Phenotype of!!Taxon Information!!Genotype Information (if known)!!Condition Information!!OMP ID!!OMP Term Name!!ECO ID!!ECO Term Name!!Notes!!Status | ||
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| + | ==Notes== | ||
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| + | ==References== | ||
| + | {{RefHelp}} | ||
| + | <references/> | ||
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| + | [[Category:Publication]] | ||
Revision as of 13:57, 22 October 2012
| Citation |
Bassford, PJ Jr, Bradbeer, C , Kadner, RJ and Schnaitman, CA (1976) Transport of vitamin B12 in tonB mutants of Escherichia coli. J. Bacteriol. 128:242-7 |
|---|---|
| Abstract |
It is known that the tonB mutation in Escherichia coli is responsible for a defect in the transport of iron chelates. These are transported by systems that involve outer membrane components. We found that tonB mutants were also deficient in the secondary, energy-dependent phase of vitamin B12 transport, although the mutants have normal levels of B12 receptors on their cell surface. In addition, tonB mutants derived from vitamin B12 auxotrophs required elevated levels of B12 for normal growth. Maltose uptake, mediated by another transport system involving an outer membrane component, was unaffected by the tonB mutation. |
| Links | |
| Keywords |
Amino Acids/metabolism; Biological Transport, Active; Enterobactin/metabolism; Escherichia coli/growth & development; Escherichia coli/metabolism; Genes; Maltose/metabolism; Mutation; Protein Biosynthesis; Receptors, Drug; Vitamin B 12/metabolism |
| edit table |
Main Points of the Paper
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Materials and Methods Used
Please list the materials and methods used in this paper (strains, plasmids, antibodies, etc).
Phenotype Annotations
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<protect>
| Phenotype of | Taxon Information | Genotype Information (if known) | Condition Information | OMP ID | OMP Term Name | ECO ID | ECO Term Name | Notes | Status |
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</protect>
Notes
References
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